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The first case was a 28-year-old man, who developed anginaatrest, followed by inferior myocardial infarction.
2
The patient was a young woman with no risk factors for coronary artery disease who had development of new-onset anginaatrest.
3
Lesion length, anginaatrest, and use of beta blockers correlated independently with slow flow in the univariate as well as in the multivariate analysis.
1
Conclusions: The absence of preinfarctionangina is more frequently observed in patients with no-reflow.
2
Earlier myocardial reperfusion may thus account for the smaller infarct size in patients with preinfarctionangina.
3
The purpose of this study was to determine the clinical significance of preinfarctionangina in the no-reflow phenomenon.
4
Background: When a myocardial infarction is preceded by angina, the infarct tends to be smaller than when there is no preinfarctionangina.
5
Methods: We compared 14 patients who had unstable angina during the week before myocardial infarction with 9 patients who had no preinfarctionangina.
Usage of unstable angina in anglès
1
Thus, sICAM-1 release is increased, especially in coronary circulation in unstableangina.
2
Women have a better prognosis than men among patients with unstableangina.
3
Significant predictors of postoperative stroke were peripheral vascular disease and unstableangina.
4
A 37-year-old male with unstableangina was admitted to our cardiovascular center.
5
However, outcomes at 6 months among patients with unstableangina differed.
6
Methods: Ninety-seven consecutive patients with unstableangina were enrolled in this single-centre study.
7
The early enhancement is common in unstableangina and could be a sign of vulnerability.
8
Main outcome measures: Admission to hospital with unstableangina, proven myocardial infarction or cardiac death.
9
We will also assess other major CHD endpoints, including revascularization and hospitalization for unstableangina.
10
Seven patients had episodes of newly developed unstableangina.
11
There was no impact on hospitalizations for unstableangina or coronary revascularization procedures with candesartan.
12
A 57-year-old man underwent myocardial revascularization for unstableangina.
13
We tested this hypothesis in a selected population of patients with unstableangina undergoing coronary angiography.
14
Fibrin deposition was mainly observed around macrophages expressing tissue factor in the patients with unstableangina.
15
There were no differences in epicardial and microvascular function between patients with stable and unstableangina.
16
The involvement of inflammation in unstableangina is suggested by the presence of activated circulating leukocytes.